Expert in Bone Health | Osteoporosis & Osteopenia Support London & Reading
OsteoPro Better Bones Programmes include:
As the Founder of Better Bones for Life™ & OsteoProSisters™, prevention and problems associated with bone health is my passion. I’m an expert in bone health, sought out for my Better Bones Talks and my OsteoPro Better Bones programmes.
Long before 1994, when osteoporosis was universally recognised as a major global public health problem and the WHO diagnostic criteria were introduced, my patients already included those with osteoporosis related problems.
Sometimes the problem was associated with primary osteoporosis: thoracic kyphosis, height loss, hip, wrist and vertebral fracture.
Sometimes with secondary osteoporosis: prolonged immobilisation following fracture, complex regional pain syndrome, cancer, another medical condition, or pharmaceutical side effects.
Since graduating in 1977, a central philosophy of my specialist clinical work has been a profound focus not on what I want individuals to achieve, but on what they themselves wish to attain.
Invariably that includes independence, return to meaningful occupations, reduced or well-managed pain, good sleep, improved mood, a much better quality of life and more laughter.
My approach is trauma-informed, positive, active, functional and future-focused.
Each client is unique and each programme a match fit. After more than 40 years of continual professional development, advanced training and certification, and practical experience from working with literally tens of thousands of people, my clinical toolkit is extensive.
An Advanced Practice Consultant Physiotherapist specialising in neurological and musculoskeletal rehabilitation, my core physical work integrates Bobath and Peto, which aligns with contemporary research on motor learning, biomechanics and neural plasticity.
My interest in science led me to certify as a gym instructor, Teacher of Tai Chi, Yoga, the Feldenkrais Method, (a neuroscience-based approach to movement), and as a certified Trainer in the Otago Falls Prevention Programme (American Physical Therapy Association), FaME Neurofitness (University of British Columbia) and Bones for Life (personally accredited by Ruthy Alon).
Additionally a Behavioural Psychologist, qualified psychotherapist, clinical hypnotherapist and story writer. A certified Trainer in Neuro-Linguistic-Programming, I’ve a deep interest in words. intonation, context, beliefs, connection, meaning, stories, mind and mood.
Throughout my career, my approach has remained consistent: helping people recover the strongest, safest and most meaningful heartfelt life possible, and when it comes to osteoporosis, there is more you can do than you realize.
Osteoporosis is a very common condition. Among the population aged over 50 years, one
in three women and one in five men will suffer a fragility fracture. 2nd Edition of the IOF Compendium of Osteoporosis 2019
Osteoporosis is a condition in which bones become less dense and more fragile.
Unfortunately that can be to the point where everyday forces that wouldn’t trouble a healthy skeleton can cause a fracture. A simple stumble, a sneeze, bending to pick something up, to move a plant pot.
The World Health Organization defines Osteoporosis as a bone mineral density T-score of −2.5 standard deviations or more below the young adult mean, measured by a DXA scan at the lumbar spine or femur [1].
But the number alone doesn’t capture what osteoporosis actually means for the people living with it — or how quietly it develops.
Bone is not static tissue. It is continuously renewed throughout life — old bone broken down by osteoclasts, new bone laid down by osteoblasts — in a process called remodelling that keeps the skeleton strong and responsive to the demands placed on it.
In osteoporosis, that balance tips: resorption outpaces formation, bones gradually lose their density and their internal structure deteriorates, and the skeleton becomes less able to bear normal loads without fracturing [2].
Because this happens without pain or warning, most people have no idea their bones have been quietly changing until a fracture, possibly after a simple trip, announces itself.
Frequency
Osteoporosis is far more common than most people realise.
The International Osteoporosis Foundation reports that worldwide, one in three women and one in five men over the age of 50 will sustain an osteoporosis-related fracture in their lifetime.
Approximately 500 million people globally are affected, and fractures occur at a rate of 70 every minute [3]
The Royal Osteoporosis Society estimates that in the UK, around 3.5 million people have osteoporosis, with many more undiagnosed [4].
Despite its scale, it is persistently undertreated: up to 80% of people who sustain a fragility fracture receive no investigation or follow-up for the underlying bone condition that caused it [3].
DXA scanning measures the mineral content of bone and remains the standard diagnostic tool, but it tells only part of the story.
Bone quality — its internal microarchitecture, the distribution of mineral, the integrity of the trabecular network — is not captured by a T-score alone.
The International Society for Clinical Densitometry notes that for premenopausal women, men under 50, and children, the Z-score — comparing bone density against age- and sex-matched peers rather than peak young adult bone mass — is the more appropriate reference [5].
Fracture risk assessment tools such as FRAX, developed in collaboration with the WHO, integrate bone density with clinical risk factors to estimate 10-year fracture probability and give a far more complete picture of what someone’s skeleton is actually at risk of [1].
References: 1, 2, 3, 4, 5
What is it?
Osteopenia means low bone mass — bone mineral density that is below the normal reference range but hasn’t yet reached the threshold for osteoporosis.
By WHO classification, that corresponds to a T-score between −1.0 and −2.5 [1].
It was originally introduced as a statistical category rather than a clinical diagnosis, and how it should be communicated, and what it should prompt, has been the subject of ongoing discussion in the clinical literature ever since.
What’s most important to understand about osteopenia is that a T-score in isolation does not determine fracture risk.
Someone can have osteopenia and be at very low risk of fracture; someone else with the same T-score and a different risk profile may be at substantially higher risk.
Risk is shaped by the whole picture: age, prior fragility fracture, family history of hip fracture, body weight, medication use, smoking, alcohol intake, and whether there are secondary conditions affecting bone metabolism.
The Bone Health and Osteoporosis Foundation and the IOF are consistent in their guidance that osteopenia should prompt a proper fracture risk assessment using FRAX — not automatic treatment, but a careful look at the full clinical picture [3, 6].
In the UK, the National Osteoporosis Guideline Group and NICE both provide frameworks for that assessment and for identifying the minority of people with osteopenia whose risk is high enough to warrant pharmacological intervention [7, 8].
For the majority of people with osteopenia, and particularly for the women I work with through OsteoProSisters, the priority is not medication — it is building bone strength, understanding what the body needs, and making informed, sustainable changes to movement, nutrition, and lifestyle.
The Dubbo Osteoporosis Epidemiology Study from the Garvan Institute found that fragility fractures and their consequences occur across a wide bone density range, including in people with osteopenia — reinforcing why proactive engagement at this stage matters so much [9].
Osteopenia is not a sentence. For many women, it is the most important signal they will ever receive about their bone health — and the best possible time to act.
References: 1, 3, 6, 7, 8, 9
What is it?
Primary osteoporosis is the most common form of osteoporosis.
Bone density loss that develops without a specific underlying disease cause. It has two distinct types, each with its own biological driver and clinical profile.
Type I — postmenopausal osteoporosis
Develops in women in the years following the menopause, driven by the decline in oestrogen.
This matters because oestrogen plays a central role in regulating bone turnover: it moderates the activity of osteoclasts, suppresses the pro-inflammatory signals that drive bone resorption, and supports the coupling between bone breakdown and bone formation that keeps the skeleton in balance.
When oestrogen levels fall, that regulation is disrupted, and bone loss — particularly in trabecular bone, which has the highest metabolic activity — can be rapid in the early postmenopausal years.
The American College of Obstetricians and Gynecologists and the Endocrine Society have both addressed the relationship between the hormonal changes of menopause and skeletal health in their clinical guidance [10, 11].
The scale of the loss in this period can be substantial, and its effect on lifetime fracture risk is significant.
Type II — Age-related or senile osteoporosis
This form of osteoporosis affects both men and women, typically from the seventh decade onwards.
It reflects the cumulative biological changes of ageing: declining osteoblast function and reduced bone formation capacity, reduced intestinal calcium absorption, changes in vitamin D metabolism, and a compensatory rise in parathyroid hormone activity that drives further resorption.
Both cortical and trabecular bone are affected, and the risk of hip fracture — with its serious implications for independence and mortality — increases substantially with age.
The American Society for Bone and Mineral Research and the Journal of Bone and Mineral Research have contributed extensively to understanding the cellular mechanisms underlying age-related bone loss and what can be done about it [12, 13].
In practice, the two types of osteoporosis, primary and secondary, frequently coexist in older women.
Postmenopausal bone loss compounded over time by the age-related changes of Type II — and the cumulative effect on the skeleton can be considerable.
Management in the UK is guided by the NOGG clinical framework and NICE guidelines, which both emphasise assessment of the full fracture risk picture rather than bone density alone [7, 8].
References: 7, 8, 10, 11, 12, 13
What is it?
Secondary osteoporosis develops when bone loss is caused or significantly accelerated by an identifiable underlying condition, medication, or clinical circumstance.
Unlike primary osteoporosis, it has a specific precipitating cause — and that distinction matters clinically, because managing secondary osteoporosis without identifying and addressing that cause is unlikely to produce lasting benefit, and may obscure the primary pathology driving the bone loss.
The range of conditions associated with secondary osteoporosis is wide.
Endocrine disorders — including hyperthyroidism, hyperparathyroidism, Cushing’s syndrome, and hypogonadism in both men and women — alter the hormonal environment that regulates bone turnover in ways that can produce significant skeletal compromise.
Malabsorptive conditions, particularly coeliac disease and inflammatory bowel disease, impair absorption of calcium and vitamin D and can cause serious bone loss even in younger people who don’t expect to be at risk.
Chronic kidney disease disrupts vitamin D activation and calcium-phosphate metabolism.
Inflammatory conditions including rheumatoid arthritis affect bone through both the disease process itself and through the treatments used to manage it [14].
Drug-induced bone loss is one of the most important and frequently overlooked aspects of secondary osteoporosis.
Long-term glucocorticoid use suppresses bone formation through its effects on osteoblast activity and raises fracture risk independently of its effects on measured bone density — NICE and the NOGG both provide specific guidance on assessing and managing glucocorticoid-induced bone loss [7, 8].
Other medications with well-documented skeletal effects include aromatase inhibitors used in breast cancer management, androgen deprivation therapy in prostate cancer, anticonvulsants, and certain antidiabetic agents.
See this article: Link: How To Strengthen Bone after Breast Cancer Treatment
The Endocrine Society and the ASBMR have both published clinical guidance on drug-related bone loss across a range of therapeutic contexts [11, 12].
Secondary osteoporosis accounts for a higher proportion of cases in men and in premenopausal women than in postmenopausal women — precisely because primary osteoporosis is less expected in these groups, so secondary causes are more likely to be at play.
A full clinical and biochemical assessment is an important step before any diagnosis of primary osteoporosis is made in younger individuals or in men.
References: 7, 8, 11, 12, 14
What is it?
Idiopathic osteoporosis is what remains when everything else has been ruled out.
It is a diagnosis of exclusion — reached only after thorough clinical and biochemical investigation has found no identifiable cause for the bone loss.
Idiopathic Osteoporosis presents in people for whom significant skeletal fragility simply cannot be explained by age, hormonal status, or any recognised secondary cause, and it requires specialist input and carefully individualised management.
It is most commonly identified in two groups: premenopausal women and men under 50 — people in whom osteoporosis is not expected, and whose presentation therefore raises important questions.
In men with idiopathic osteoporosis, research published in PubMed-indexed literature has identified reduced serum IGF-1 alongside histomorphometric evidence of impaired osteoblast function and reduced bone formation — suggesting that the problem lies in the bone-building side of the remodelling cycle rather than in excess resorption [15, 16].
In premenopausal women, studies published in the Journal of Bone and Mineral Research have documented profound deficits in bone microarchitecture — thinner cortices, reduced trabecular connectivity, lower volumetric bone mineral density — alongside variable bone remodelling patterns, and paradoxical IGF-1 relationships that differ from the pattern seen in men, suggesting the underlying mechanisms are not the same across sexes [17, 18].
This is not a homogeneous condition with a single explanation.
Idiopathic osteoporosis also occurs in children and adolescents — juvenile idiopathic osteoporosis — typically presenting around the pubertal growth spurt with vertebral and long bone fractures.
This is distinct in its natural history and management from adult presentations and requires specialist paediatric assessment.
The clinical challenge is that the evidence base for management in younger adults with idiopathic osteoporosis is considerably less developed than for postmenopausal osteoporosis.
The ISCD and the clinical research literature both emphasise that standard protocols designed for postmenopausal bone loss are not always appropriate or well-evidenced in this population, and that management requires specialist assessment, comprehensive evaluation of bone microarchitecture where available, and genuinely individualised clinical thinking [5].
References: 5, 15, 16, 17, 18
Understanding what osteoporosis is — and which type applies — is the essential foundation.
But for the people living with it, the diagnostic category is rarely the most pressing reality.
What matters most is what osteoporosis actually does: to the skeleton, to how the body moves and feels, to confidence and independence, to sleep and mood and daily life.
Osteoporosis doesn’t exist in isolation. It sits within — and interacts with — a wider musculoskeletal and physiological picture.
Reduced bone strength increases fracture risk; fear of fracture restricts movement; reduced movement contributes to muscle loss; muscle loss compromises balance and reduces the mechanical loading that bone depends on.
Pain, deconditioning, anxiety, and loss of confidence form a cycle that the structural diagnosis alone doesn’t begin to address.
Understanding those interconnections — and working with the whole person rather than only the bone density reading — is what genuinely integrated bone health care looks like and what I offer at my practice.
The following sections address prevention, elevated osteoporosis risk and the specific problems associated with osteoporosis: fragility fracture risk: fragility fractures, falls and falls risk, persistent pain, sarcopenia, spinal deformity, and the particular needs of people with elevated risk.
Each is addressed in terms of what it is, what it means for the person living with it, and how it is managed in my practice.
The reality is that most osteoporosis services begin after bone loss has already occurred, after osteopenia has been identified, and that is frequently after a fragility fracture has disrupted confidence, mobility and independence.
Yet we know that many of the foundations for lifelong skeletal health are established much earlier, throughout childhood and adolescence.
This is during the years when bone is developing most rapidly and remains highly responsive to physical loading, nutrition and environmental influence [1,2,3].
Healthy Bones Australia states clearly that calcium, vitamin D and weight-bearing exercise all play essential roles in bone health throughout life, although their relative importance changes across childhood, adulthood and later life [1].
This understanding and my desire to work upstream from the problems associated with Osteoporosis, sits behind the development of my Osteo-Pro: ProBone Activate programme.
This is a specialist health promotion and prevention programme designed to support parents of children and young people under the age of sixteen. With a particular focus on those aged approximately three to fourteen.
The programme recognises that community, parents and family environments strongly influence children’s behaviour, their movement habits, outdoor activity, exposure to nature, healthy nutrition, health beliefs and long-term behaviour patterns during critical years of skeletal development [2,4,5,6].
Research consistently shows that childhood and early adolescence represent important windows for building stronger bones and improving long-term skeletal resilience [1,2,3,7].
Weight-bearing and impact-based activities including running, jumping, climbing and active play stimulate bone adaptation during growth, while inactivity reduces these opportunities [1,7,8].
The Canadian Paediatric Society advises that supporting healthy bones in childhood involves regular physical activity, adequate calcium intake, sufficient vitamin D and reducing prolonged sedentary behaviour [2,9].
Bone health is also increasingly being understood within a wider environmental and public health context.
Research published in JAMA Network Open identified associations between exposure to residential green space and improved bone mineral density in young children, highlighting the importance of active outdoor environments during early development [10].
Other studies show that family members remain among the strongest influences on young people’s health knowledge and behaviours, reinforcing the importance of parent-focused education and early intervention [4,5,11].
At the same time, research suggests that many adolescents and young adults still possess poor knowledge of osteoporosis and bone health, despite the importance of these early years [12].
The Osteo-Pro: Better Bones Programme therefore has a programme designed to help families build stronger foundations earlier — long before osteoporosis and fragility fracture conventionally become part of the conversation.
For many of the women I work with through Better Bones For Life, the most important moment isn’t the diagnosis of osteoporosis — it’s the period before it.
A family history of fragility fracture, the early signs of hormonal changes, irregular menstrual cycles, hot flashes, night sweats, vaginal dryness, sleep disruptions, mood changes, and “brain fog.
In addition a recognition that there may be risk factors, long-term medication use, low body weight, years of inadequate nutrition, sedentary work, lack of exercise that osteogenic and promotes bone formation, development, and the stimulation of new bone tissue.
These are all signals that bone health needs attention now, not after a diagnosis of Osteopenia, or something breaks.
Healthy Bones Australia describes exercise as playing a vital role across the entire lifespan: building bone in youth, maintaining it through adulthood, and preserving the mobility and balance that prevent falls in later life [6].
The Garvan Institute created the Know Your Bones tool specifically because awareness and early engagement are fundamental to prevention — understanding your own risk is the first step towards doing something about it [4].
And yet Osteoporosis Canada’s data confirm that fewer than half of people at elevated risk are meeting basic physical activity or nutritional recommendations.
This means the gap between what we know and what people are actually doing is large, and consequential [7].
Effect
Being told your bones are at risk lands differently for different people.
Some women feel energised by it — this is information they can act on, and they want to.
Others feel anxious, confused, or quietly frightened, particularly if the clinical conversation that delivered the information was brief and the pathway forward unclear.
Some avoid finding out at all.
The period of elevated risk that precedes a diagnosis or a first fracture is not a passive waiting room — it is the time when the choices being made every day are shaping the bone health of the next decade.
Research from the Garvan Institute’s Dubbo Study makes clear that fractures, and their consequences, occur across a wider age range and broader bone density profile than was previously understood.
This reinforces why acting before a threshold is crossed matters so much [3].
For the women I work with in this space, what they need is not a leaflet.
They need real information, a programme that makes sense for their life, and support from someone who understands both the clinical picture and what it actually means to live in a body that feels less certain than it used to.
That is what I provide with my programme.
Management
Healthy Bones Australia is direct about what works: bones need specific types of exercise to respond — weight-bearing impact, progressive resistance training, and high-challenge balance work.
General activity, things most people do on an average day, helps overall health, but it isn’t enough to build or maintain bone on its own [6].
For women with osteopenia or identified risk factors, the foundations are bone-targeted movement, nutritional optimisation — adequate calcium, protein, and vitamin D — lifestyle awareness, and sustained, supported engagement with their own bone health.
Osteoporosis Canada’s 2023 guidelines lead with exercise as their primary recommendation, before everything else, naming balance and functional training alongside progressive resistance work as the first-line approach [7].
The Garvan Institute notes that healthy lifestyle behaviours across adulthood — diet and consistent progressive weight-bearing exercise — are the most important contributors to bone health in later life [4].
The Royal Osteoporosis Society’s Strong, Steady and Straight consensus statement confirms that exercise for bone health needs to be targeted, progressive, and individually tailored to be effective [19].
In my OsteoPro Bone Better Bones programme, that is exactly what I offer — evidence-based, individually tailored movement, education, and lifestyle support for women who are ready to take an active role in their bone health, and who deserve far more than a brief clinical encounter and a standard information sheet.
References: 3, 4, 6, 7, 19
A fragility fracture is a fracture that happens from a force that wouldn’t break a healthy bone — a fall from standing height, a stumble, sometimes something as unremarkable as a sneeze or turning over in bed.
Fragility fractures are the most serious clinical consequence of osteoporosis, and what the research tells us is sobering.
The Garvan Institute of Medical Research’s Dubbo Osteoporosis Epidemiology Study — one of the longest-running osteoporosis studies in the world — established that every low-trauma fracture significantly increases the risk of another, with the highest risk window in the first five years after the initial fracture [1, 2].
It also showed that premature mortality rises following fractures across the skeleton, not just hips as was previously assumed [1].
And critically, half of all fragility fractures occur in people whose bone density doesn’t even reach the diagnostic threshold for osteoporosis — which is why looking at the whole fracture risk picture matters so much more than a single number from a DXA scan [3].
Effect
For many people, a fragility fracture is the moment they realise their bones have been quietly losing strength for years.
It is frightening, often painful, and frequently accompanied by a sudden, disorienting loss of what felt possible.
A hip fracture can be life-changing — many people who were independent before do not recover the same level of independence afterwards.
Vertebral (spine) fractures bring their own slower, cumulative burden: persistent back pain, height loss, changes in posture, and the altered mechanics that follow multiple collapsed vertebrae.
Wrist and shoulder fractures take away the daily tasks that feel most personal — getting dressed, cooking, driving, managing alone.
And across all of it, the fear of another fracture often does more damage than the fracture itself — because when we fear we reduce and can even stop movement, and it is movement that bones and muscles need most.
Management
A fragility fracture is a clinical event that demands a full response — not just treatment of the broken bone, but a genuine plan to prevent the next one.
The Garvan Institute has made clear that proper management following fracture could substantially reduce re-fracture risk — and yet the majority of people who sustain a fragility fracture still receive no bone health investigation or follow-up [4].
The evidence for what actually works is now strong.
The LIFTMOR randomised controlled trial at Griffith University, led by Watson, Weeks, Weis, Harding, Horan and Beck, showed that a short, twice-weekly programme of high-intensity resistance and impact training measurably improved bone density and physical function in postmenopausal women with low bone mass — safely, and with over 90% adherence [5].
Healthy Bones Australia identifies weight-bearing impact exercise, progressive resistance training, and challenging balance work as the evidence-based triad that bones respond to across the lifespan [6].
Osteoporosis Canada’s 2023 clinical practice guidelines lead with exercise — balance, functional training, and progressive resistance work — as the primary intervention for fracture prevention [7].
In my practice, recovery from a fragility fracture is always a whole-person process: rebuilding physical strength, movement confidence, and the sense that it is safe — and worthwhile — to move again.
References: 1, 2, 3, 4, 5, 6, 7
Falls and fractures go together in osteoporosis — you can’t address one without addressing the other.
Healthy Bones Australia reports that half of all falls in older adults happen at home, in familiar surroundings, doing everyday things [6].
The British Geriatrics Society describes falls in older people not as individual events with a single cause, but as a syndrome — the result of multiple things happening at once: reduced muscle strength, slower reactions, changes in balance and spatial awareness, altered gait, the effects of medication on blood pressure or alertness, and the accumulated impact of other health conditions [8].
In someone with osteoporosis, each of those factors carries more weight precisely because the consequence of falling is so much greater than it would be for someone with healthy bone.
Effect
Living with an elevated risk of falling does something quiet but significant to a person’s life. Behind all actions, movement behaviours and movement practices can lie a low level fear of more falls and more fractures.
Activities that were previously automatic start to require thought.
Social situations that involve uneven ground, unfamiliar spaces, or low lighting get quietly avoided.
The fear of falling — quite apart from whether a fall actually happens — is associated with reduced physical activity, social withdrawal, and a gradual narrowing of life.
And that narrowing matters, because it weakens the very muscles and balance systems that would have protected against a fall in the first place.
When osteoporosis and sarcopenia are both present — a combination now recognised as osteosarcopenia — the risk is substantially compounded.
A systematic review and meta-analysis published in Osteoporosis International confirmed that osteosarcopenia is associated with significantly higher rates of falls, fractures, disability, and mortality compared with either condition alone [9].
Management
The evidence for exercise in falls prevention is as strong as any evidence in musculoskeletal health — and the key is that balance training needs to be genuinely challenging to work.
Healthy Bones Australia is clear that the nervous system only adapts when it is pushed: that means balance tasks that change direction, require concentration, and are progressively made harder over time [6].
The British Journal of Sports Medicine supports progressive resistance and balance training as the primary approach to maintaining neuromuscular function and reducing falls in older adults [10].
Griffith University’s LIFTMOR research programme showed that high-intensity bone-targeted exercise — the foundation of the Onero programme — builds both bone and muscle strength safely across a wide age range, including people with established bone loss [5].
Osteoporosis Canada’s 2023 guidelines place balance and functional training at the top of their exercise recommendations, more than twice weekly [7].
In every programme I run, falls risk and the psychological impact of trauma, and movement confidence are addressed directly — they are not an afterthought.
References: 5, 6, 7, 8, 9, 10
Osteoporosis is often called a silent disease because it causes no pain or symptoms until something breaks.
But once a fracture has occurred — particularly in the spine — pain can become a persistent, complex, and deeply disruptive part of daily life.
Vertebral (spine) compression fractures are the most common source of ongoing back pain in osteoporosis, and their pain rarely behaves simply.
Acute fracture pain may ease over weeks, but for many people it gives way to something longer-lasting: pain driven by the altered mechanics of a changed spine, by the neurological sensitisation that develops when pain is present for a sustained period, and by the anxiety and muscle tension that chronic pain almost always brings with it.
The structural change and the pain experience do not map neatly onto one another.
Many people with significant vertebral changes have modest symptoms; others with limited imaging findings are in severe, function-limiting pain. Both are real, and both deserve to be taken seriously [11].
Effect
Persistent pain from osteoporotic fracture changes life quietly and incrementally.
Sleep is disrupted. Movement becomes something to manage rather than something that happens naturally.
Everyday things — carrying a bag, turning over in bed, sitting through a meal, reaching a shelf — become calculations.
The isolation that accompanies unrelenting pain is well documented: depression and anxiety are common in people living with chronic musculoskeletal pain, and in the context of osteoporosis they carry particular weight — because the pain comes alongside fear of further fracture, anxiety about movement, and often a profound sense of loss about what the body used to be able to do [12].
The gap between how someone looks from the outside and how they are actually managing is often enormous, and that invisibility is its own exhausting burden.
Management
Effective pain management in osteoporosis means addressing the whole picture — not just the structural source of the pain but the nervous system that is amplifying it, and the emotional experience that is shaping it.
Movement is central, not despite the pain but as part of managing it.
Back extensor strengthening is one of the most consistently evidenced physical interventions in osteoporotic vertebral fracture — it improves spinal support, gradually reduces kyphotic posture, and reduces pain over time [13].
The ESCEO and the IOF both support individually tailored exercise as a core part of management across all osteoporosis presentations, including those complicated by pain and fracture history [14].
Pain neuroscience education — helping people understand why pain persists, what the nervous system is doing, and why the scan result doesn’t explain everything — changes how people relate to their pain, and that shift makes a real difference to what they feel able to do.
Psychological support addressing trauma, anxiety, depression, sleep, and fear of movement is not a luxury or an add-on — in persistent pain, it is essential.
In my practice, pain is always understood as a whole-person real experience, not a purely structural problem with a structural solution.
References: 11, 12, 13, 14
Sarcopenia is the progressive loss of muscle mass, strength, and function that comes with ageing.
Sarcopenia and osteoporosis are not separate problems that happen to coincide. They are deeply connected.
Muscle contraction is one of the most important stimuli for bone formation.
When muscles pull on bone through movement and loading, bone responds by maintaining and building its structure.
When muscle mass and strength decline, that stimulus is reduced, and bone loss accelerates.
The relationship runs in the other direction too — pain, fracture, and restricted movement reduce the physical activity that muscle depends on, so the two conditions tend to spiral together rather than develop in isolation.
When both are present simultaneously — now recognised clinically as osteosarcopenia — the ESCEO and the IOF have both highlighted that the combined impact is greater than either condition alone.
Shared risk factors include ongoing and possibly reducing inactivity, low protein and vitamin D intake, hormonal changes, and chronic low-grade inflammation make this combination common, particularly in older women [9, 14].
Effect
The practical consequences of sarcopenia alongside osteoporosis are deeply felt in daily life.
As muscles get weaker through inactivity, getting up from a chair takes effort.
Stairs require thought. Carrying shopping, maintaining balance on uneven ground, moving quickly when something unexpected happens — all of these become harder.
Loss of physical strength changes what feels possible, and with it, what people choose to do.
Social life narrows. Activities get dropped. Independence, which most people only notice when it starts to slip, is incrementally affected.
A systematic review published in Osteoporosis International confirmed that osteosarcopenia is associated with a significantly elevated risk of falls, fractures, disability, and mortality compared to either condition in isolation [9].
Beyond the physical, there is the emotional dimension — the loss of a body that felt capable, the anxiety that comes with recognising increasing fragility, and the dependency that muscle weakness can begin to create in relationships.
These things deserve to be part of the clinical conversation, not left unaddressed alongside the physical prescription.
Management
The good news — and it is genuinely good news — is that muscle responds to the right exercise at any age.
Progressive resistance training is the most robustly evidenced intervention for sarcopenia, and its effects on bone and muscle are not two separate outcomes: they happen together.
The LIFTMOR trial at Griffith University demonstrated that twice-weekly high-intensity resistance and impact training produced significant improvements in bone density, muscle strength, and functional capacity in postmenopausal women with low bone mass — safely, with high adherence throughout [5].
The LIFTMOR-M trial extended those findings to older men [15].
Healthy Bones Australia places progressive resistance training — with progressive increases in load — alongside weight-bearing impact and challenging balance work as the essential framework for both bone and muscle health [6].
Osteoporosis Canada’s 2023 guidelines recommend progressive resistance training targeting back extensors and core muscles more than twice weekly as a primary clinical recommendation [7].
Nutrition matters equally: muscle protein synthesis requires adequate protein and vitamin D, and the ESCEO has identified nutritional optimisation alongside exercise as the foundation of managing osteosarcopenia — not one or the other, but both together [14].
References: 5, 6, 7, 9, 14, 15
When vertebrae fracture and partially collapse — as they commonly do in osteoporosis, often without a dramatic event and sometimes without the person even realising what has happened — the spine changes shape.
Progressive anterior wedging of the vertebral bodies produces the forward rounding of the upper back that is widely associated with osteoporosis: thoracic kyphosis.
Lateral displacement can produce or worsen scoliosis.
These changes are not cosmetic. They reflect real structural compromise of the spinal column, with consequences that ripple through the whole body — how it moves, how it breathes, how it carries itself through daily life.
Research documents that even vertebral fractures that produce no immediate symptoms are associated with measurable declines in pulmonary function as the thoracic space reduces [16].
Effect
As the kyphosis increases, the body’s centre of gravity shifts forwards.
Maintaining an upright position becomes effortful, placing sustained demand on the muscles of the back and lower limbs — muscles that are often already weakened by sarcopenia.
Gait changes and balance becomes less secure. The risk of further falls rises.
Height loss — which can amount to several centimetres over multiple fractures — is distressing in ways that go beyond the physical: it changes how people see themselves, how clothes fit, how they move through a world designed for a body they used to have [17].
Restricted breathing — because the rib cage has less room to expand — is rarely discussed in clinical consultations but is very much part of the lived experience, as is the early fullness and reduced appetite that comes from abdominal compression in pronounced kyphosis.
And the visible change in posture itself carries an emotional weight: for many women it becomes a marker of age and fragility that they did not choose, affecting confidence, self-presentation, and willingness to engage in social life.
Management
Back extensor strengthening is one of the most well-evidenced physical interventions in vertebral osteoporosis, improving the dynamic support of the spine, gradually reducing kyphotic posture, and reducing pain over time [13].
Healthy Bones Australia and Osteoporosis Canada’s 2023 guidelines both identify targeted exercise for back extensors and core as a specific clinical priority in this population [6, 7].
Balance and proprioceptive work addresses the gait and stability consequences of altered spinal alignment — directly relevant to falls risk.
The Onero programme, developed by Professor Belinda Beck at Griffith University through the LIFTMOR trial and developed further through the STOP FRACTURE! study, has demonstrated that high-intensity resistance and impact training builds bone and improves muscle strength safely even in those with existing vertebral fractures, under appropriate supervision [5, 18].
In my practice, the psychological impact of postural change — on confidence, identity, and social engagement — is never treated as peripheral. It is part of the whole-person picture that good bone health care must address.
References: 5, 6, 7, 13, 16, 17, 18
⚠ Any exercise or physical activity undertaken without prior consultation with a qualified healthcare professional is done at your own risk. The information provided is for general guidance only and does not constitute medical advice. We recommend that you consult a physiotherapist before starting any new exercise or activity and book a consultation to arrange an individualised programme tailored to you.
Recovery: What’s almost always missed?
Yes it’s Trauma and here’s the science bit.
Definition
Trauma is the psychological and physiological response to an event or series of events that is experienced as overwhelming, exceeding an individual’s capacity to cope, and resulting in lasting effects on functioning and wellbeing.
What about genes?
Trauma does not change the DNA sequence, but it can alter gene expression through epigenetic mechanisms (e.g. DNA methylation, stress-hormone regulation), influencing pain sensitivity, inflammation, and stress reactivity. There is strong evidence that early life stress and trauma are associated with long-term changes in the HPA axis, increasing vulnerability to chronic pain and heightened symptom response. This helps explain why individuals with similar structural conditions can present very differently in pain, function, and recovery.
More
Trauma is not only biological; the experience of injury, diagnosis, or loss of identity (e.g. athlete, performer) can itself be psychologically traumatic.
Individuals may develop acute stress or PTSD-type responses following accidents or even clinical consultations, particularly where events are sudden, threatening, or life-altering. This can amplify pain, disrupt recovery, and alter engagement with rehabilitation through fear, avoidance, and heightened threat perception.
If you want a higher quality of life, are open to making a behaviour change and working in fresh, new ways for better future health, do give me a call.
Section 1 References
Section 2 References
a) Prevention and Early Bone Health
B) Elevated risk, and the specific problems associated with Osteoporosis
Wherever you are in your bone health journey, we start with a 20 minute conversation, and we meet online or over the phone.
Perhaps you’ve been reading about osteoporosis and realize you have some risk factors, a family member with osteoporosis and could develop osteoporosis.
Some individuals call when they have just received a diagnosis of osteopenia or osteoporosis and aren’t sure what it means or what to do next.
Some are parents who want to give their children the best possible foundation for lifelong bone health.
Others have been living with bone-related pain, fracture history, or increasing falls risk for some time and are looking for an approach that goes further than anything they have encountered so far.
Wherever you are starting from, the first step is a conversation. I offer a free, pre-booked twenty-minute introductory call — online, at a time that suits you — where we can talk through your current situation, what has or hasn’t worked before, what you are hoping to achieve, and which of my programmes is likely to be the most appropriate fit.
There is no obligation, and you will leave the call with a clear sense of possible next steps, realistic expectations, and an honest picture of what working together would involve, including costs.
A full online assessment is also available — details of what this covers and how it works are set out on a separate page — and this forms the foundation of any individualised programme recommendation.
Each OsteoPro distinct programmes, is designed for a specific stage of the bone health journey. Getting you into the right one matters. That is what the initial conversation is for.
Appointments are available online across the UK, in Reading, in London, and through selected home visits by arrangement.
We use cookies to improve your experience on our site. By using our site, you consent to cookies.
Manage your cookie preferences below:
Essential cookies enable basic functions and are necessary for the proper function of the website.
These cookies are used for managing login functionality on this website.
Statistics cookies collect information anonymously. This information helps us understand how visitors use our website.
Google Analytics is a powerful tool that tracks and analyzes website traffic for informed marketing decisions.
Service URL: policies.google.com (opens in a new window)
You can find more information in our Cookie Policy and Privacy Policy.